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Latest comment: 1 year ago by OhanaUnited in topic Peer review 1

WikiJournal Preprints
Open access • Publication charge free • Public peer review

WikiJournal User Group is a publishing group of open-access, free-to-publish, Wikipedia-integrated academic journals. <seo title=" Wikiversity Journal User Group, WikiJournal Free to publish, Open access, Open-access, Non-profit, online journal, Public peer review "/>

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Article information

Submitting author: Jens Staal[a][i] 
Additional contributors: Wikipedia community

See author information ▼
  1. Ghent University
  1. jens.staal@irc.vib-ugent.be

Plagiarism check

[edit source]

Pass. Report from WMF copyvios tool: only trivial overleap with other sources such as "and motor proteins, and transcription factors". T.Shafee(Evo﹠Evo)talk 22:46, 8 February 2023 (UTC)Reply

Editorial comments

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Comments by Andrew Leung ,
These editorial comments were submitted on , and refer to this previous version of the article

Similar to WikiJournal Preprints/MALT1, I would like to see the following points to be improved before sending it to peer reviewers:

  1. The evolution and species distribution covered the history aspect from an evolutionary perspective. It should provide a brief history on how it was discovered (and by whom). See WikiJournal of Medicine/A history of coronaviruses for example.
  2. The bullet-point sentences in "Functions" section should be organized into paragraphs based on commons themes.

Again, you can take a look at WikiJournal of Science/RIG-I like receptors and WikiJournal of Science/The TIM barrel fold as examples.

OhanaUnitedTalk page 17:33, 15 May 2023 (UTC)Reply

Peer review 1

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Review by Daniel Krappmann , Helmholtz Zentrum München
These assessment comments were submitted on , and refer to this previous version of the article

The summary on CARD-CC looks good and should be informative to the readers. As I don’t have much time to edit online or so, I simply suggest to add something to the section on function:

… CBM complex assembly results in recruitment of TRAF6 to MALT1 and downstream activation of NF-κB transcriptional activity and expression of pro-inflammatory cytokines (REF1). In addition, CBM complex formation activates MALT1 protease function, which modulates immune and inflammatory responses (REF2). The different CARD-CC family…

I would add these Refs for clarity:

REF1: O’Neill, T.J., T. Seeholzer, A. Gewies, T. Gehring, F. Giesert, I. Hamp, C. Graß, H. Schmidt, K. Kriegsmann, M.J. Tofaute, K. Demski, T. Poth, M. Rosenbaum, T. Schnalzger, J. Ruland, M. Göttlicher, M. Kriegsmann, R. Naumann, V. Heissmeyer, O. Plettenburg, W. Wurst, and D. Krappmann, TRAF6 prevents fatal inflammation by homeostatic suppression of MALT1 protease. Science Immunology, 2021. 6(65): p. eabh2095. DOI: 10.1126/sciimmunol.abh2095.

REF2: Jaworski, M. and M. Thome, The paracaspase MALT1: biological function and potential for therapeutic inhibition. Cell Mol Life Sci, 2016. 73(3): p. 459-73. DOI: 10.1007/s00018-015-2059-z.

OhanaUnitedTalk page 18:39, 7 September 2023 (UTC)Reply