Progress and Prospects in Parkinson's Research/Pathogenesis
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This page is an introduction to the pathogenesis of Parkinson's disease. It will introduce the consensus as it is currently emerging about how the disease starts and develops at the cellular level.
The topics and subpages will elaborate on these ideas and reference the evidence for them.
Genetics
[edit | edit source]"Defects in SNCA [Gene ID: 6622] have been implicated in the pathogenesis of Parkinson disease."[1]
"A heterozygous guanine to cytosine mutation at the first base of codon 389 in MAPT [GeneID: 4137] is identified in a case of juvenile frontotemporal dementia with parkinsonism."[2]
Related pages
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Subpages:
See also
[edit | edit source]References
[edit | edit source]- ↑ RefSeq, Mar 2009 (11 August 2014). "SNCA synuclein, alpha (non A4 component of amyloid precursor) [ Homo sapiens (human) ]". Bethsda, Maryland USA: NCBI. Retrieved 2014-08-14.
- ↑ GeneRIFs 8. (11 August 2014). "MAPT microtubule-associated protein tau [ Homo sapiens (human) ]". Bethsda, Maryland USA: NCBI. Retrieved 2014-08-14.
Further reading
[edit | edit source]External links
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